Ashrafi, Kaveh
Barber, Diane L
Bernstein, Harold S.
Black, Brian L
Blanc, Paul D
Boushey, Homer A
Broaddus, V Courtney
Brown, James K
Caughey, George H
Chapman, Harold A
Charo, Israel F
Chatterjee, Kanu
Chuang, Pao-Tien
Clyman, Ronald I
Conklin, Bruce R
Coughlin, Shaun R
Derynck, Rik M
Dobbs, Leland G
Eisner, Mark D
Engel, Joanne N
Erle, David J
Fahy, John Vincent
Farese, Robert V
Fielding, Christopher J
Fielding, Phoebe
Fineman, Jeffrey R
Glantz, Stanton A
Grossman, William
Hawgood, Samuel
Ingraham, Holly A
Jan, Lily Y
Kan, Yuet W
Kane, John P
Kornberg, Thomas B
Kurtz, Theodore W
Kwok, Pui-Yan
Lazarus, Stephen C
Malloy, Mary J.
Martin, Gail R
Matthay, Michael A
Mcdonald, Donald M
Mikawa, Takashi
Minor, Daniel L
Mostov, Keith E
Nadel, Jay A
Ordahl, Charles P
Pitas, Robert E
Reiter, Jeremy F.
Rosen, Steven D
Shaw, Robin M.
Sheppard, Dean
Simpson, Paul C
Stainier, Didier Y. R.
Wang, Rong
Weiner, Orion D
Weisgraber, Karl H
Weiss, Arthur
Weiss, Ethan J
Werb, Zena
Wiener-Kronish, Jeanine
Young, William L

CVRI Scientists

Harold S. Bernstein, M.D., Ph.D.
Associate Professor of Pediatrics

Research Interests:
Cell cycle regulation, myocyte biology, cardiac muscle, stem cells

Summary:
Each year, more than 900,000 people in the U.S. experience a heart attack, and about 500,000 die from complications of heart failure. Heart failure occurs when heart muscle cells are damaged and the heart is unable to meet the demands placed on it by the body. Unlike other organs, the heart is unable to repair itself. We seek to develop new ways of stimulating heart muscle repair or producing replacement heart muscle cells to be used for repair, thereby treating or avoiding heart failure.

CVRIHead